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(**A**). We observed in particular that SARS-CoV-2, in contrast with SARS-CoV (panels A, B), was able to rapidly reach a very high viremia titer for a limited time, followed by a strong decrease of viremia (**C**). This viremia decrease was accompanied by a drop of cTnI serum level, and a second viremia increase was observed. On the other hand, the other two patients showed a moderate increase of cTnI serum level, followed by a continuous decrease (**D**).](jcm-09-02681-g001){#jcm-09-02681-f001}

![Immunohistochemistry for ACE2 in a control subject. Human lung tissue was immunostained with antibodies specific for ACE2 (original magnification: × 200). (**A**) Non-infected human lung tissue. (**B**) Lung tissue of a patient infected with SARS-CoV-2. (**C**) Lung tissue of a patient infected with SARS-CoV. Absence of cytoplasmic staining of human lung tissue with the ACE2 antibody (original magnification: × 200).](jcm-09-02681-g002){#jcm-09-02681-f002}

![Western blot analyses for ACE2 expression in the human lung tissues. Lung tissues were collected at autopsy from control subjects (*n* = 6) and patients with SARS-CoV (*n* = 7) and SARS-CoV-2 (*n* = 5) infection. Tissue lysates were blotted on nitrocellulose membranes. Western blot analyses revealed that, in human lung tissues, ACE2 levels were high in the control samples, whereas in SARS-CoV samples only weak ACE2 was detectable. SARS-CoV-2

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Shoemaster Qs 10 02 |LINK| Crack 👹

Shoemaster Qs 10 02 |LINK| Crack 👹

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